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The researchers stated, “When GLP-1 drugs create a calorie-deficient state, the brain appears to increase the activity of AgRP neurons so that fat is lost or regulated,” adding, “This implies that GLP-1 drugs operate through mechanisms that had not been recognized until now.” This study is significant because it presents a new principle: GLP-1 drugs may go beyond simple appetite suppression to drive sustained fat reduction through brain neural networks
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