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Ca2+ influx through L-type Ca2+ channels and Ca2+-induced Ca2+ release regulate cAMP accumulation and Epac1-dependent ERK 1/2 activation in INS-1 cells
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Without the Clostridium butyricum and Bifidobacterium infantis supporting strains, the GLP-1 stimulating effects are reduced, because the synergistic butyrate production pathway is missing
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